By 4% which may as well be experimental error, right? The mice could have very well just lived longer due to increased interest/handling/measurement of their welfare.<p>This seems insignificant when compared with other selector genes such as H63D and C282Y, which could potentially double or halve subjects life span depending on environmental factors.
I would really like to see the end of aging and by that I mean the end of age related disease, dying from ALS or Alzheimer's disease (which both are by and large age related diseases) is not nice. My mom has ALS and it is tough.<p>I don't see much other hope in curing these diseases tbh, it feels like the research is going nowhere so I am somewhat excited about longevity research and glad that someone is pursuing it. If I didn't become a software developer, I would like to work in that field
I don't quite understand the premise of longevity research.<p>Isn't there a concept similar to "no free lunch" in biology? Infinite, cancer-free lives sounds just too good to be true. Something sounds off about that.
If I were a mad scientist I'd attempt to transplant bat mitochondria into mice.<p>Bats must have lots of adaptations in their mitochondrias to how much calories they burn.
We're diminishing biodiversity to our own peril and disadvantage.<p>The primary driver of biodiversity loss and deforestation is animal agriculture.<p>Act accordingly.<p><a href="https://www.pnas.org/doi/10.1073/pnas.2204892120" rel="nofollow noreferrer">https://www.pnas.org/doi/10.1073/pnas.2204892120</a><p><a href="https://en.wikipedia.org/wiki/Holocene_extinction" rel="nofollow noreferrer">https://en.wikipedia.org/wiki/Holocene_extinction</a><p><a href="https://dothemath.ucsd.edu/2023/08/ecological-cliff-edge/" rel="nofollow noreferrer">https://dothemath.ucsd.edu/2023/08/ecological-cliff-edge/</a>